F&N Digest
Extension Foods and Nutrition, Cooperative Extension Service, Kansas State University

May/June 1996

Food Safety
Questions and Answers About Bovine Spongiform Encephalopathy, Mad Cow Disease (BSE)
Healthy Food Preparation
Modified Recipe: Chocolate Milk Shake
You Asked It
You Asked It -- Rapid Response Center Q/A
Limited Resource
Malnutrition and Cognitive Development
Need for Food Assistance Increasing
Trends in Food and Alcohol Consumption Away From Home
Nutrition and Health
Olestra, A Dieter's Dream or Dilemma?
Obesity And Your Genes
International No Diet Day (INDD)
Fat Intake Continues to Drop But Veggies and Fruits Still Low in US Diet
Tips for Working Moms on Handling and Storing Breastmilk
Grain Products Will Be Fortified with Folic Acid to Prevent Birth Defects

Questions and Answers About Bovine Spongiform Encephalopathy, Mad Cow Disease (BSE)
What is BSE?
BSE is an extremely rare, chronic degenerative disease affecting the central nervous system of cattle. BSE is not present in the U.S., but has been identified in the United Kingdom and in a few other countries.

Has BSE ever been detected in the U.S.?
No cases of BSE have been detected in the U.S. In fact, over the last nine years, USDA has completed 2,660 tests on brains from cattle that have shown any possible neurological problems. All tests were negative.

What steps has USDA taken to prevent BSE from entering the U.S.?
To prevent BSE from entering the United States, USDA's Animal and Plant Health Inspection Service (APHIS) has taken a number of steps, including:

  • APHIS has banned the importation of live ruminants and ruminant products from countries where BSE is known to exist. Since 1991, there has been a voluntary ban in place on the use of rendered products from adult sheep in animal feeds.
  • In 1986, APHIS established a program for BSE surveillance in the U.S. and provided specialized training for 250 APHIS veterinarians who conduct field investigations involving suspicious symptoms. APHIS veterinary pathologists and field investigators have received training from British counterparts for diagnosing BSE.
  • More than 60 veterinary diagnostic laboratories throughout the United States are participating in a BSE Surveillance Program (initiated in May 1990) along with the National Veterinary Services Laboratories in Ames, IA.
  • APHIS veterinarians are tracing 499 head of cattle imported from Great Britain between 1981 and 1989 (before the ban on imports went into effect) to check their health status. As of January 22, 1996, 106 imports are known to be alive; 341 are known to be dead; and eight have been exported. APHIS is trying to locate the remaining 34, but based on the age of the animals, they are assumed to be dead. The animals that are alive are monitored regularly, and no signs of BSE have been found.

How widespread is BSE and what has the British government done to control its spread?
Between 1986 and September 1995, an estimated 156,000 head of cattle in more than 32,000 herds were diagnosed with BSE in Great Britain. The number of cases peaked in December 1992, and has declined to less than one-third of that rate and is still declining. A series of precautionary steps taken in Great Britain have resulted in the decline in newly reported cases. For example, in July 1988, Great Britain banned the feeding of ruminant-derived protein to ruminants. Currently fewer than 300 cases are occurring per week.

Is it true that you can get Creutzfeldt-Jakob Disease (CJD), a human brain disease, from eating beef from cattle with BSE?
Experts in Great Britain are concerned there may be a link between BSE and a small group of 10 cases of CJD. However, the British government's scientific advisors have noted that there is no direct evidence of a link. If there is a risk, it likely would be linked to consumption of brain or spinal cord from infected animals. Scientific evidence indicates that beef (meat) and milk do not present a risk as there is no evidence the agent that causes BSE is present in meat and milk.

What is the incidence of CJD?
CJD occurs at consistent rates of approximately one case per million people per year around the world, including in nations where BSE has never occurred. In fact, BSE is found only in limited areas. CJD also occurs at a consistent rate among vegetarian groups and meat eaters alike, which would indicate that its cause is not meat consumption. Scientific evidence suggests several possible causes of CJD, including genetic predisposition and certain surgical procedures.

Where was BSE first detected?
BSE was first identified in 1986 in Great Britain. While it has been found in a few other countries, the vast majority of BSE cases have been concentrated in the United Kingdom. BSE is similar to other animal degenerative brain diseases that include Chronic Wasting Disease in elk and deer and Scrapie which has been known for more than 100 years to affect sheep.

Is BSE a virus or a bacterial infection?
The scientific evidence suggests that BSE is not a viral or bacterial infection. Rather, it seems to point to a protein material or "prion" as the cause of the disease.

How do cattle get BSE?
Scientists conclude that cattle may get the disease from eating protein in feed that was probably contaminated with a spongiform disease agent. Scientific evidence indicates that BSE does not spread from cattle to cattle or from cattle to other species by physical contact.

What are the symptoms of BSE?
Cattle with BSE have coordination problems and are very nervous. In the advanced stages, infected cattle stand away from the rest of the herd and exhibit severe muscular twitching and weight loss. (KP)

Source: NCBA Advisory March 26, 1996


Modified Recipe: Chocolate Milk Shake

Chocolate Milk Shake (Original)
1 1/2 medium scoops chocolate ice cream
1 1/2 cups whole milk

Combine ingredients in blender. Makes one 16 oz. serving.

Chocolate Milk Shake (Modified)
1 1/2 bananas
3 tsp Unsweetened cocoa powder
1 cup Nonfat skim milk
1 T Maple Syrup (or sugar or other sweetener)

Combine ingredients in blender. Makes one 16 oz. serving. (Substitutions: choose your favorite sweetener, just don't use too much! Non-fat chocolate syrup can be used instead of cocoa powder, but you will be adding calories.)

Nutrition per serving
  Original Modified
Calories 438 306
Fat 23 g 2 g
Cholesterol 83 mg 5 mg
Sodium 254 mg 151 mg
Percent of Calories from Fat 47% 6%
Total Dietary Fiber 1 g 5 g

(RB)


You Asked It! -- Rapid Response Center Q/A
Clarification: In regards to the question about testing a candy thermometer in the last issue of F&N Digest (March/April 1996), a clarification is needed: An altitude adjustment is necessary when calibrating the thermometer using boiling water. At sea level, water boils at 212 F (100 C), however, if you live at a higher elevation, water will boil at a lower temperature. This must be taken into consideration when calibrating the thermometer. Use the boiling point of water for your altitude:

Altitude (Ft.) Boiling Point
0 212 F (100 C)
1000 210 F (99 C)
2000 208 F (98 C)
3000 206 F (97 C)

NOTE: Thanks go out to Dr. Aramouni (Ext. F&N) and Dr. Boyle (Ext. Animal Science) for this clarification

Q. Is the "floating egg" test a good test for egg freshness?
A. Yes and no. In a general way, it is because as an egg ages, moisture and carbon dioxide leave through the pores in the shell and are replaced by air. Thus, the larger the air pocket, the older an egg is, and an egg with a larger air pocket will float compared to an egg with a smaller air pocket.

However, this is not a good test because it will not tell you if an egg is bad, only that it is older compared to a fresh egg.

Q. What is the shelf-life of corn meal?
A. Corn meal may be stored on the shelf for over one year in a tightly closed container. (Complete cupboard, refrigerator, and freezer storage charts are available at the Rapid Response Center.)

Q. What is the difference between refined and unrefined oil?
A. Refined oil has been treated with an alkali to remove free fatty acids. Refined oil will have a longer shelf life because free fatty acids are much more susceptible to rancidity in the unrefined oil.

Q. Do you have any nutritional information for rattle-snake?
A. The best that the Rapid Response Center can do is provide information about SNAKE, general, raw: 100g contains 92 cal, 18 g protein, 2 g fat, and 1 g carbohydrate. (NOTE: Nutritional information for the following wild foods can be obtained at the Rapid Response Center: FROG, raw; GRUB, larva, raw; INSECT, silkworm, raw; RAT, cooked or raw; SNAILS, small; and TREE ANTS, whole.)

Q. Are the RDA's (Recommended Dietary Allowances) from 1989 the most current?
A. Yes. They are the basis for the Daily Values that you see on food labels.

Q. Why does regular cream cheese have less calcium than lowfat cream cheese?
A. This is a nutrient density question. Calcium is found in the non-fat portion of the cream cheese. If fat is removed, is still the same amount of calcium, but it becomes a higher percentage of the total.

Q. I know that you cannot make Jell-O with fresh pineapple. What about fresh kiwi?
A. No. Each contains an enzyme (Pineapple _ bromelin, Kiwi _ actinidin) that retards gelation, thus the Jell-O will never set.

Q. What does the date on an egg carton mean?
A. The date on a carton of eggs is the sell by date. Refrigerated eggs are still good for 4-5 weeks after this date.

Q. What are Craisins (C-Raisins)?
A. Dried cranberries. (RB)


Malnutrition and Cognitive Development
A recent study of childhood malnutrition and mental development provides further evidence of the impact of poor diet on cognitive development.

In a long-term study of four Guatemalan villages from 1969-1989, children and pregnant women received one of two nutritional supplements: Atole, a high protein gruel, and Fresco, a sweet drink which contained no protein and only one-third of the calories of Atole. In follow-up studies of the participants 20 years later, researchers gave the villagers a battery of literacy, vocabulary and reading comprehension tests, a general knowledge exam, a math test and a standard nonverbal intelligence test.

Participants who had received the Atole supplement scored much higher on cognitive tests than did those given Fresco. The poorest Atole subjects performed as well on the tests as wealthier local peers, eliminating income differences as performance factors. However, all of the villages participating in the study were extremely poor, and children did not perform as well as children from middle income families in more affluent areas in Guatemala. The researchers point out that proper nutrition cannot totally compensate for the negative effects of poverty on mental growth.

Federal food assistance programs such as Food Stamps, WIC, and the School Breakfast Programs help prevent childhood malnutrition and contribute to cognitive development. Studies have shown, for example, that children participating in School Breakfast have significantly higher standardized achievement test scores than eligible non-participants. (MP)

Source: CNI Feb. 16, 1996


Need for Food Assistance Increasing
Participation in food pantry programs and the Food Stamp Program (FSP) is on the rise.

According to a recent survey of 29 cities conducted by the U.S. Conference of Mayors, the number of requests for emergency food assistance is increasing, particularly among families with children and the elderly. Unfortunately, the cities surveyed indicated they are unable to meet the increased demand for food since resources for emergency food facilities increased by only 1.5% in 1995. Almost all officials surveyed predicted that requests will increase in 1996.

Food Stamp participation has increased dramatically between 1989 and 1992 from 59% of those eligible to 74%. A report by the Food and Consumer Service attributed the increase to both the economic recession as well as better access to food stamp offices and more effective outreach efforts.

In 1992, the FSP reached 95% of all eligible children under five and over 80% of children. Participation of the eligible elderly increased 5%. (MP)

Source: CNI Feb. 16, 1996


Trends in Food and Alcohol Consumption Away From Home
According to the 1992 Consumer Expenditure Survey, American households spent 38% of their food dollar on food away from home and 46% of their alcohol dollar on alcohol consumed outside the home. Consumers who spent the greatest share of their food dollar on food away from home were in the highest income quintile, under age 25, or living alone. Sales at eating and drinking places were up 134% between 1980 and 1993, with a 48% increase in the number of employees working at these establishments.

Factors influencing the decision to eat out include the increasing numbers of women in the labor force, the trend toward more one-person households, price competition among restaurants, and the interest in restaurants that offer some type of entertainment. (MP)

Source: Family Economics and Nutrition Review 8:4 1995.


Olestra, A Dieter's Dream Or Dilemma?
Note: This is the second article about olestra. See March/April 1996 issue for more information.

Olestra, the fake fat produced by Procter and Gamble and recently approved by the Food and Drug Administration, is only allowed at the present time in potato chips, tortilla chips and crackers. According to some, it's a dieter's dream because olestra, marketed under the trade-name Olean, is not absorbed out of the intestine and into the blood stream and thus provides no calories. Yet, Olean has the mouth-feel of fat. It makes you think you're eating a high fat food when you're not.

Olestra has been languishing in the wings for years awaiting FDA approval. Costing some $200 million to develop, research and market, it is likely the costliest diet product to come onto the market.

You would think that with FDA's approval the issues regarding its safety and effectiveness would be settled. Such is not the case. A vocal minority call it a "public health expert's nightmare: an uncontrolled experiment on 200 million people using an unsafe food additive."

Everyone agrees that olestra provides that all-important sensory attribute of fat smoothness. We eat much more by a food's ability to satisfy our senses than because of its cost-effectiveness or nutrition profile. What experts don't agree on is its proven safety. And the argument may go on for years because the subtle effects of olestra on health may take years to develop and then document. The two major issues concern the carotenoids and the small intestine.

Carotenoids

"Don't eat olestra along with carotenoid-rich foods such as carrots, broccoli, greens, cantaloupe, and apricots." So goes the advice about consuming beta-carotene-rich foods with olestra. The problem is that olestra picks up the fat-soluble vitamins, A, D, E and K, including carotenoids and removes them from the body. This means that you don't want to eat carrot sticks and olean potato chips at the same meal.

But beta-carotene is only one of hundreds of carotenoids found naturally in plants. For example, lycopene, a caratenoid found in tomatoes, is associated with protecting men against prostate cancer and lutein and zeaxanthin, carotenoids found in green leafy vegetables like spinach, are associated with preventing macular degeneration, an irreversible deterioration of the retina and a cause of blindness in older adults. Those most concerned about olestra's safety contend that we are just learning about the ways that carotenoids and other phytochemicals protect health. They contend that even selective fortification with the fat-soluble vitamins is likely not enough especially when viewed with the disappointing results from the beta-carotene supplementation trials. These trails showed that beta-carotene was not protective against lung cancer in smokers. Thus eating fruits and vegetables appears to be far more health protective than consuming selected nutrient supplements. Just eight grams of olestra a day (16 chips worth) can lower blood lutein levels by 20 percent and blood lycopene by 60 percent.

Digestive discomfort

Both olestra and high-fiber diets can cause diarrhea, gas and abdominal cramping. In one of the studies, just eight grams of olestra in 16 potato chips caused diarrhea in some people. Twenty grams, as in a two-ounce single-serving bag, led to diarrhea in a third of the volunteers. The volunteers did not develop a tolerance for olestra whereas people eating more fiber-rich foods have noted that after several days they adapt to the extra fiber. Fiber stimulates the growth of more "good" bacteria in the intestines thereby reducing symptoms.

Questionable weight loss

Finally, some question whether or not olestra can help reduce our collective waistlines. Despite all the money spent on dieting products, the obesity rates in both children and adults rose a third during the 80's rather than falling as you might expect. People falsely assume they can indulge if they eat low-fat or fat-free foods.

FDA has been asked to place a warning label on foods containing olestra because of the unknown but possible link between increased risks for cancer, heart disease and blindness. The Federal Trade Commission has likewise been asked to require the same warning for all advertisements for olestra products. No doubt, the olestra story and controversy will continue to unfold. (MC)

Source: Karstadt, Myra, & Stephen Schmidt. Olestra, Procter's big gamble. CSPI Nutrition Action Healthletter. 23(2):4-5. March 1996.


Obesity And Your Genes
Researchers are getting a handle on why it is so easy for some people to get fat. It could be their genes.

Leptin, a fat cell hormone

A major breakthrough come in December 1994 when researchers at Rockefeller University in New York discovered the ob gene in a strain of overweight mice. Ordinarily, fat cells produce a protein called leptin. Leptin is a hormone that travels around in the blood stream and signals the brain that the body has stored enough fat. In leptin-deprived mice due to an absent or defective gene, the mice overeat, become inactive and grow fat. The startling finding was that when the fat mice were injected with leptin, they ate less, slimmed down and burned calories faster than before. Even normal-weight mice lost body fat due to leptin.

Could these findings apply to humans? Unlike mice, humans do not have a mutated ob gene. In fact, obese people not only have enough leptin but may actually have 20 to 30 times more than lean people. So the researchers have looked for another answer such as: Could brain cells lack the receptors that would be necessary to receive the message? Researchers have now found this leptin receptor in the brain. The next step is to find out if these receptors are faulty in some obese people. If so, then perhaps a drug can be designed that will improve their receiving ability for leptin.

But before hopes get too high, this leptin explanation may be responsible for only a small percentage of obesity. Drug stimulation might not help those with severely defective or missing receptors. Perhaps gene therapy would be necessary but that solution could be decades away. In the meantime, the various labs will be looking for gene mutations in hundreds of obese people.

Now GLP-1, (glucagon-like peptide-1)

An even newer finding is GLP-1, a "stop-eating" signal. This appetite-suppressing substance is found in brain and intestine tissue. Some scientists suspect that GLP-1 may be at least one of the end results of leptin's effects. GLP-1 acts separately in both the brain and the digestive tract. In the brain, it triggers feelings of satiety; in the digestive tract it slows stomach emptying and increases blood levels of insulin.

Perhaps this substance will turn out to be even more important than leptin. Certainly drug companies will be working overtime to develop weight-loss medications. Whoever comes up with an effective and safe weight-control drug is destined to reap huge rewards.

Dexfenfluramine, a new appetite-control drug

In the meantime, dexfenfluramine (to be marketed as Redux) is being tested after narrowly receiving approval by an FDA advisory panel last November. It will likely be available as a prescription medication by the end of the year. Dexfenfluramine has long been available in Europe but U.S. approval has been slow because of concerns about possible brain damage. The drug suppresses the craving for carbohydrates because it stimulates more serotonin, a neurotransmitter produced in the brain that affects both appetite and mood. Researchers are worried about its long-term health effects.

Currently, weight-loss drugs (fenfluramine and phentermine) can only be prescribed for short periods. When people stop taking them, they generally regain the lost weight.

Despite all of these promising developments, most obesity experts agree that genes are only partly responsible for obesity. Epidemiologic data suggest that much of our obesity is environmentally based, i.e., we eat too much and exercise too little. Pills will not solve that problem but they may be able to help control appetite.

Before World War II, obesity rates were half what they are today (now 1 of every 3 adults is obese). Obviously, genes don't mutate that quickly. It takes many generations to make even small changes. What was different back before the war was that people ate less fat and more fiber and exercised more. No matter what, it appears that we will still need changes in our eating and exercise patterns in order to maintain or improve weight. (MC)

Source: Garnett, Leah R. Is Obesity All in the Genes? Harvard Health Letter. 21(6):1-3. April 1996.


International No Diet Day (INDD)
There is a growing movement towards size acceptance, that is the idea that it is no sin to be fat and that people can be healthy and live health-promoting lifestyles even though their bodies do not conform to our cultural fantasies. Certainly, overweight may be harmful to your health but it is not a moral issue nor a sign of a weak character. Unfortunately, obesity is more socially unacceptable than most other physical diversities.

This year International No Diet Day is being celebrated May 6. Over 100 organizations world-wide have banned together to promote No-Diet Day. Some organizations in this country who are involved are the National Association to Advance Fat Acceptance (NAAFA) [916-443-0303 or P.O. Box 188620, Sacramento, CA 95818]; the Association for the Health Enrichment of Large People (AHELP) [1-800-368-3468, Ext. 501 and ask for Bernice]; and Largesse, the Network for Size Esteem [P.O. Box 9404, New Haven CT 06534-0404, Phone/fax 203-787-1624]. Karen Stimson is the Co-director of Largesse and a contact if you are interested in being more involved in No Diet Day either this year or in the future.

Another excellent way to get information is to use the INDD web site (http://www.texmed.org/has/pin/dan/ndd1.asp). It has information on the origin of INDD, its goals, people to contact, a calendar of events, sample press releases, fact sheets, and other resources. If you want the INDD Handbook, send $1 and a self-addressed #10 envelope to Largesse at the mailing address above. If you see someone with a light blue ribbon on May 6th, they are observing No-Diet Day.

A sign of the times is the realization by professionals as well as consumers that present unrealistic expectations for a person's ability to reach idealized "perfect body" goals should be challenged. Leaders in this movement think that those of us working in health promotion should be accepting of and sensitive to size diversity. We should promote body satisfaction and the achievement of realistic and attainable health goals without regard to weight change.

To this end, they propose the following Tenets:

  • Human beings come in a variety of sizes and shapes. We celebrate this diversity as a positive characteristic of the human race.
  • There is no ideal body size, shape, or weight that every individual should strive to achieve.
  • Every body is a good body, whatever its size or shape.
  • Self-esteem and body image are strongly linked. Helping people feel good about their bodies and about who they are, can help motivate and maintain healthy behaviors.
  • Appearance stereotyping is inherently unfair to the individual because it is based on superficial factors which the individual has little or no control over.
  • We respect the bodies of others even though they might be quite different from our own.
  • Each person is responsible for taking care of his/her body.
  • Good health is not defined by body size; it is a state of physical, mental, and social well-being.
  • People of all sizes and shaped can reduce their risk of poor health by adopting a healthy lifestyle. (MC)

Fat Intake Continues to Drop But Veggies and Fruits Still Low in US Diet
Americans are eating less fat as a percentage of total calories, according to a new U.S. Department of Agriculture nationwide food survey.

Data from the first year of the three year survey -- "What We Eat in America" -- found that Americans consumed 33 percent of calories from fat in 1994, continuing a downward trend. In the late 1970's, Americans consumed 40 percent of calories from fat . According to a 1989-1991 survey, Americans consumed 34 percent of calories from fat.

Even with this decrease, however, two-thirds of adult Americans still eat more than the 30 percent recommended limit for fat calories. The survey also found that nearly as many are consuming more than the recommended limit for saturated fat.

In addition, survey participants in 1994 reported a total calorie intake about six percent higher than 17 years ago. Thirty percent of the men and 45 percent of the women reported that they rarely engaged in vigorous exercise. This may help explain why participants reported weights averaging 11 to 12 pounds heavier. They also reported heights averaging one inch taller. Based on these measurements, one in three adults was overweight in 1994 compared with one in five in the earlier survey.

Less than one-third of American adults meet the recommendation to consume five or more servings of fruits and vegetables per day. And Americans still consume low amounts of dark green and deep yellow vegetables, despite recommendations to do otherwise.

Consumption of grain-based products is on the rise. For example, between the late 1970's and 1994, Americans increased their consumption of ready-to-eat cereals by 60 percent and their consumption of snacks such as crackers, popcorn, pretzels, and corn chips by 200 percent. Consumption of grain mixtures -- such as lasagna and pizza -- increased 115 percent during that time.

The data are based on personal interviews during 1994 with some 5,500 Americans of all ages across the country. They were asked to recall their food intake for two days. The figures are average intakes, so many Americans either fall above or below the average.

The survey results identified some of the following trends:

  • About 20 percent more fruit or fruit juice was consumed in 1994 than 17 years ago, but still nearly half of the population reported eating no fruit on a given day.
  • Milk consumption decreased by 16 percent for children under five since the late 1970's.
  • Consumption of soft drinks rose by 23 percent and consumption of noncitrus juices -- such as mixes with a grape or apple base -- skyrocketed more than 300 percent.
  • Among adults, neither men nor women had average zinc or magnesium intakes that met the Recommended Dietary Allowance. Women's intakes for iron, vitamin B6, calcium and vitamin E were also below the RDA.
  • In 1994, more than half of Americans ate away from home on the day in question (see related article in this issue), accounting for about one-quarter of their total calorie and fat intakes. And the number of women and young children eating away from home has increased by about 50 percent since the late 1970's.

When USDA's three-year survey concludes in early 1997, they expect to have data on between 15,000 and 16,000 Americans, including low-income individuals, children and the elderly. The data are used to make public policy decisions related to food safety, food fortification, food assistance and nutrition education programs and also for research, food production and marketing and many other areas. (PP)

Source: Agricultural Research Service, U.S. Department of Agriculture Beltsville Human Nutrition Research Center


Tips for Working Moms on Handling and Storing Breastmilk
A working mom can continue to provide breastmilk for her baby after she returns to work. If the infant is in child care, both mom and child care provider will need to work together to safely handle the breastmilk.

The first responsibility comes for the mom. She needs to be sure to express and store the breastmilk safely:

  • Wash her hands with soap and water before she starts.
  • Express milk into a clean container, either glass or plastic.
  • Put single feeding-size portions into bottles for storage. This could be two to three ounces for the very small infant or more as the baby grows. Freeze some one-half to one ounce servings to have on hand for days when the baby many want a little more to eat.
  • Label and date the bottles so that oldest ones can be used first.
  • Store in the refrigerator if it will be used within 3 or 4 days, otherwise freeze.
  • Frozen breastmilk will keep up to three months in the freezer portion of the refrigerator or up to six months in a separate freezer.
  • When transporting breastmilk from work to home or home to child care provider: Use a cooler with ice or ice pack. If frozen breastmilk thaws, and it is kept refrigerated, it can be used within 24 hours.

Some tips for the child care provider for serving breastmilk include:

  • Thaw frozen breastmilk by running cold water over the container, increasing the warmth of the water gradually while shaking the container.
  • Test to be sure the breastmilk is body temperature before serving.
  • Avoid boiling or heating in the microwave. Excess heating can destroy the protective substances in breastmilk. Microwaving can also create "hot spots" which could burn the baby.
  • Once breastmilk has thawed, it can spoil more easily. Keep it cold and use within a day or two. Do not refreeze.
  • Make sure the bottle and nipple are clean. Discard any leftover breastmilk.

(PP)

Sources: Children's Nutrition Research Center. Nutrition and Your Child. Winter 1996. Guidelines for Collecting and Handling Breastmilk, Kansas Cooperative Extension and Kansas Lean Preschool Taskforce, in press.


Grain Products Will Be Fortified with Folic Acid to Prevent Birth Defects
U.S. food manufacturers will add the nutrient folic acid to most enriched breads, flours, corn meals, pastas, rice and other grain products to reduce the risk of neural tube birth defects in newborns, as a result of action taken on February 29, 1996, by the Department of Health and Human Services and the Food and Drug Administration.

Folic acid, or folate, reduces the risk of neural tube birth defects such as spina bifida when consumed in adequate amounts by women before and during early pregnancy. Spina bifida is a common disabling birth condition resulting from failure of the spinal column to close. Spina bifida and other neural tube birth defects affect approximately 4,000 pregnancies each year in the U. S. Genetics also plays a role in development of these neural tube defects. Many genetic factors affect how we utilize folic acid, including an aberrant gene which produces an enzyme which makes it necessary for some women to consume more folic acid than normal to prevent neural tube defects.

The Public Health Service recommended in 1992 that all women of child-bearing age consume 0.4 milligrams of folic acid daily to reduce their risk of giving birth to children with neural tube defects. As part of the Public Health Service strategy to achieve that goal, the FDA fortification rule is aimed at increasing folate intakes.

Under the new FDA rules, specified grain products will be required to be fortified with folic acid at levels ranging from 0.43 milligrams to 1.4 mg per pound of product. These amounts are designed to keep daily intake of folic acid below 1 mg, because intakes above that amount may mask symptoms of pernicious anemia, a form of vitamin B12 deficiency which primarily affects older people. If untreated, pernicious anemia can lead to severe permanent nerve damage.

Because over half of all pregnancies are unplanned and because these defects occur in the developing fetus before most women know they are pregnant, it is important that all women of child-bearing age consume the needed 400 micrograms (0.4 mg) of folic acid daily.

In addition to fortified grain products, it is recommended that women should obtain this amount through food sources, such as leafy dark green vegetables, citrus fruits and juices, and lentils; dietary supplement (such as a multivitamin); or a combination of these sources.

Foods required to be fortified are: enriched bread, rolls, and buns; all enriched flour including bromated and self-rising flours; enriched corn grits and corn meals; enriched farina and rice; and all enriched macaroni and noodle products including vegetable macaroni, vegetable noodle, and non-fat milk macaroni products. In addition, breakfast cereals can add folic acid up to 400 mcg per serving.

Also under the new rules, manufacturers will be allowed to make claims on the labels that the fortified products contain folate or folic acid and that adequate intake of the nutrient has been shown to reduce the risk of neural tube birth defects.

These final rules on fortification will become effective Jan. 1, 1998. FDA will review any new data concerning optimum fortification levels for folic acid and will consider adjusting the level of fortification if the data substantiate the need. (PP)

Sources: FDA News Release, February 29, 1996. Community Nutrition Institute. Nutrition Week, March 15, 1996.


K-State Research and Extension is a short name for the Kansas State University Agricultural Experiment Station and Cooperative Extension Service, a program designed to generate and distribute useful knowledge for the well-being of Kansans. Supported by county, state, federal and private funds, the program has county Extension offices, experiment fields, area Extension offices and regional research centers statewide. Its headquarters is on the K-State campus, Manhattan.